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MTHFR, Liver Toxicity, and Fibromyalgia

By January 11, 2013No Comments

Environmental exposures are now often involved in the inability of normal detox pathways to keep pace. Genetic defects such as Methyl Tetra-Hydrofolate Reductase Enzyme (MTHFR) defect, and Phase 2 liver defects, lead to increased exposure within the matrix or extra-cellular space. This space is where all the hormones, immune cells, and neurotransmitters communicate with one another. With the extra toxins present communication is interrupted. Communication is hampered and the long term effects can include endocrine dysregulation or deficiency, immune distraction and eventually anergy, and neurotransmitter depletion. Testing as offered through standard labs such as Labcorp, and Sonora Quest, are able to determine deficiencies and imbalances. Treatment initially restores normal function and balance while supporting detox pathways. As toxins are removed we can see normal production and function restored to the immune, endocrine, and neurotransmitter systems. Detox pathways are enhanced through the liver, kidneys, colon, and skin to help remove toxins at a faster rate reducing the total load.

Read on for the more articles about Fibromyalgia…>>>Adrenal Fatigue and Fibromyalgia